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This study is to explore a GSH-responsive AP probe-net, which can be activated by the abundant GSH in ATC cells to generate reactive alkoxyamine, and the formed alkoxyamine can trap AP sites and block the APE1 activity, leading to ATC cell death but less adverse effects towards normal cells

The trial is registered with the University Hospital Medical Information Network (UMIN) Clinical Trials Registry (UMIN000011118)

To ensure blinding integrity, assessors were instructed not to discuss treatment details with patients, and patients were advised not to disclose their treatment to assessors

(Bethesda) 29 (4), 242249

Further, this data suggests that rather than buffering oxidative stress to prevent liver damage, GSH synthesis is crucial for maintaining low NRF2 levels and sustaining lipid production by the liver